嗜肺軍團菌尿液檢測試紙

廣州健侖生物科技有限公司

（廣州健侖生物科技有限公司是集研制開發、銷售、服務于一體的優良企業,公司產品涉及臨床快速診斷試劑、食品安全檢測試劑，違禁品快速檢測，動物疾病防疫檢測試劑，免疫診斷試劑、臨床血液學和體液學檢驗試劑、微生物檢驗試劑、分子生物學檢驗試劑、臨床生化試劑、有機試劑等眾多領域，同時核心代理Panbio、FOCUS、Qiagen、IBL、CORTEZ、Fuller、Inbios、BinaxNOW、LumuQuick、日本富士、日本生研等多家有名診斷產品集團公司產品，致力于為商檢單位、疾病預防控制中心、海關出入境檢疫局、衛生防疫單位,緝毒系統，戒毒中心，檢驗檢疫單位、生化企業、科研院所、醫療機構等機構與行業提供*、高品質的產品服務。此外，本公司還開展食品、衛生、環境、藥品等多方面的第三方檢測服務。）

主要用途：用于檢測尿樣中嗜肺軍團菌血清型1抗原，以支持軍團菌感染的診斷。

產品規格：20T/盒

存儲條件：2-30℃

嗜肺軍團菌尿液檢測試紙

我司還提供其它進口或國產試劑盒：登革熱、瘧疾、西尼羅河、立克次體、無形體、蜱蟲、恙蟲、利什曼原蟲、RK39、漢坦病毒、深林腦炎、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、化妝品檢測、食品安全檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。

歡迎咨詢

歡迎咨詢2042552662

【產品介紹】

二維碼掃一掃

【公司名稱】 廣州健侖生物科技有限公司
【】    楊永漢 
【】 
【騰訊 】 2042552662
【公司地址】 廣州清華科技園創新基地番禺石樓鎮創啟路63號二期2幢101-3室
【企業文化】

6.攝取與分泌神經遞質
神經膠質細胞既能攝取，又能分泌神經遞質。如：腦內星形膠質細胞能攝取谷氨酸與γ－氨基丁酸兩種遞質，以消除這兩種遞質對神經元的持續作用；同時又可通過星形膠質細胞的代謝，將這兩種遞質再轉變為神經元可重新利用的遞質前體物質。此外，星形膠質細胞還能合成并分泌血管緊張素原、前列腺素、白細胞介素以及多種神經營養病毒子等生物活性物質。神經膠質細胞通過對神經遞質或生物活性物質的攝取、合成與分泌。而發揮其對神經元功能活動的調節作用。
7.吞噬作用
此外，小膠質細胞和星形膠質細胞有吞噬作用，能吞噬有病變的神經
1. 神經元急性壞死（紅色神經元，red neuron）：急性缺血、缺氧，急性中毒或感染可引起神經元的死亡，表現為神經元核固縮，胞體縮小變形，胞漿尼氏小體（Nissl body）消病毒，HE染色胞漿呈深伊紅色，稱為紅色神經元(red neuron)。如細胞壞死后的酶性分解過程繼續發展，則可導致細胞核溶解消病毒，殘留細胞的輪廓或痕跡稱為鬼影細胞（ghost cell）。由缺血引起的紅色神經元zui常見于大腦皮質的錐體細胞和小腦蒲肯野（Purkinje）細胞。
2. 單純性神經元萎縮（simple neuronal atrophy）:神經元慢性漸進性變性以至死亡的過程稱為單純性神經元 萎縮。又稱神經元的慢性病變。病變特點表現為神經元胞體縮小，核固縮而無明顯的尼氏小體溶解，一般 不伴炎癥反應。晚期可伴明顯膠質細胞增生。
3. 中央性Nissl小體溶解（ce病毒al chromatolysis）：為一種可逆性變性，常由病毒感染，維生素B缺乏及神經 元與軸突斷離等病毒素所致。病變表現為神經元腫脹、變圓、核偏位，胞漿中央的尼氏小體崩解，進而溶解 消病毒，或僅在細胞周邊部有少量殘余，胞漿著色淺而呈蒼白均質狀。
4. 神經元胞質內包含體形成（i病毒ancyto-plasmic inclusion）：神經元胞質內包含體形成可見于某些病毒感染 和變性疾病等，其形態、大小和著色不同，分布部位也有一定規律，如Parkinson病的黑質，藍斑等處的神 經細胞中的Lewy小體（圖14-2）；狂犬病時海馬和腦皮質錐體細胞中的Negri小體，分別對這些疾病具有診 斷意義。此外，神經元胞漿中出現脂褐素多見于老年人。

6. Uptake and secretion of neurotransmitters
Glial cells can not only uptake, but also neurotransmitters. Such as: astrocytes in the brain can take up glutamate and γ-aminobutyric acid two neurotransmitters in order to eliminate the two neurotransmitters on the sustained effect; at the same time through the astrocyte metabolism , Converting these two neurotransmitters into neurotransmitter precursors. In addition, astrocytes also synthesize and secrete biologically active substances such as angiotensinogen, prostaglandins, interleukins and various neurotrophic viruses. Glial cells through the neurotransmitter or bioactive substances uptake, synthesis and secretion. And play its role in the regulation of neuronal functional activity.
7. phagocytosis
In addition, microglia and astrocytes have phagocytosis that engulfs diseased nerves
1. Neuronal Acute Necrosis (red neuron): Acute ischemia, hypoxia, acute poisoning or infection can cause neuronal death, manifested as neuron nuclear condensation, cell body deformation, cytoplasmic Nissl small Body (Nissl body) virus, HE staining cytoplasm dark red, known as red neurons (red neuron). If the process of enzymatic decomposition after necrosis continues, the nuclei will dissolve the virus and the outline or trace of residual cells will be called the ghost cell. Red neurons caused by ischemia are most commonly found in the pyramidal cells of the cerebral cortex and in Purkinje cells of the cerebellum.
2. Simple atrophy of nerve cells (simple neuronal atrophy): The process of chronic progressive degeneration and death of neurons is called simple neuronal atrophy. Also known as neuronal chronic disease. Lesions characterized by shrinkage of neuronal somatic cells, nuclear pyknosis and no significant Nissl body lysis, generally do not have inflammatory reactions. Late with obvious glial cell proliferation.
3. Central Nissl body lysis (ce virus al chromatolysis): a reversible degeneration, often caused by viral infection, vitamin B deficiency and neurons and axons off the other toxins. Lesions showed neuronal swelling, rounding, nuclear deviation, the central cytoplasmic Nissl body disintegration, and then dissolve the virus, or only a small amount of residual cells in the periphery, the pale cytoplasm was pale homogeneous shape .
4. Neuronal cytoplasmic inclusion body formation (i virus ancyto-plasmic inclusion): Neuronal cytoplasmic inclusion bodies can be seen in some viral infections and degenerative diseases, the shape, size and coloring of different distribution sites also have a Such as Lewy bodies in neurons at the substantia nigra and locus coeruleus of Parkinson's disease (Figure 14-2); Negri bodies in the hippocampus and pyramidal cells of rabbits at the time of rabies are diagnostic for these diseases, respectively . In addition, the presence of lipofuscin in neuronal cytoplasm is more common in the elderly.